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[DOI] [PMC free article] [PubMed] [Google Scholar] 30.Meinhardt J., Radke J., Dittmayer C., Franz J., Thomas C., Mothes R., Laue M., Schneider J., Brnink S., Greuel S., Lehmann M., Hassan O., Aschman T., Schumann E., Chua R.L., Conrad C., Eils R., Stenzel W., Windgassen M., Rler L., Goebel H.H., Gelderblom H.R., Martin H., Nitsche A., Schulz-Schaeffer W.J., Hakroush S., Winkler M.S., Tampe B., Scheibe F., Krtvlyessy P., Reinhold D., Siegmund B., Khl A.A., Elezkurtaj S., Horst D., Oesterhelweg L., Tsokos M., Ingold-Heppner B., Stadelmann C., Drosten C., Corman V.M., Radbruch H., Heppner F.L

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The failure of mitochondrial steroidogenesis in GCs can result in aberrant oocyte development, especially when estrogen levels in follicular fluid are low and progesterone levels are high, affecting proper meiosis and late cleavage of oocytes (80, 102, 103)

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